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The hypothesis is that the fundamental healing mechanisms BPC-157 promotesangiogenesis, collagen synthesis, anti-inflammationare universal and would therefore apply to the damaged annulus fibrosus of a disc

Paradoxically, while PPAR- activation enhances lipid storage, it concurrently improves hepatic insulin resistance and inhibit inflammatory responses, oxidative stress, and endoplasmic reticulum stress in this key metabolic organ [69,70,71,72]

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