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MPO and hypochlorous acid (HOCl) expression were significantly upregulated in NSC-34 cells transfected with the human SOD1 G93A gene, leading to the activation of the MPO/HOCl signaling pathway

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Some researchers suggest that it inhibits an enzyme variant related to COX-2 in the brain, which produces pain and inflammation signaling molecules, thus reducing pain sensation

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We propose an important contribution of de novo ser/gly synthesis pathway activation to RT responses by supplying ATP, nucleotides, and antioxidants to support the survival and/or recovery of cancer cells upon RT