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Studies have demonstrated that the induction of ICD by chemotherapeutic agents, particularly anthracyclines, is accompanied by phenomena such as the unfolded protein response within the endoplasmic reticulum (ER) and the generation of reactive oxygen species (ROS)
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The mechanism-based rationale for concerns is strong, however, especially if applied to settings in which these peptides are used on a long-term basis [2, 24]
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TMAO has been proven to aggravate CVD by promoting atherosclerosis and thrombotic risk and has also been reported to be correlated with other cardiometabolic diseases such as non-alcoholic fatty liver disease and chronic renal disease [6,7,8]
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