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J Cancer Epidemiol
Through phosphorylation inhibition of 4E-BP1, mTORC1 can increase ODC translation and promote polyamine synthesis (Rousseau et al., 1996)
Additionally, IL-4-induced Arg1 expression was further upregulated by choline deprivation and Lico E treatment, reinforcing the hypothesis that CTL1 inhibition fosters a neuroprotective anti-inflammatory phenotype promoting A degradation ( The role of choline metabolism in inflammation extends beyond microglia
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Standard IGF-1 remains important for in vivo studies where IGFBP interactions are part of the biological system under investigation
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