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Research has revealed that TGF- stimulates the canonical Wnt signaling pathway in a p38-dependent manner by downregulating the expression of the Wnt antagonist Dickkopf-1 ( Figure 3 details the molecular cascade of the canonical Wnt pathway, highlighting how its activation by TGF- leads to -catenin stabilization and subsequent transcription of pro-fibrotic genes, underscoring its significance as a potential therapeutic target

The Journal of clinical psychiatry

Tetraspanins on the Surface of Schistosoma Mansoni Are Protective Antigens Against Schistosomiasis

Review important medication safety information

Diabetic kidney disease

Graphical abstract Similar content being viewed by others Introduction The prevalence of neurodegenerative diseases (NDDs), including Parkinsons disease (PD) and Alzheimers disease (AD), has been increasing rapidly due to the aging global population, which has placed a significant burden on healthcare systems [1,2,3]