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Their metabolic pathways diverge from ours in ways that can make a miracle compound in one species become a dudor worse, a dangerin another

PMCID: PMC7281326

Concerning GSTLs, based on proteome analyses and on the presence of an N-terminal predicted targeting sequence, AtGSTL2 is clearly a plastidial protein and it may also be present in peroxisomes as shown by GFP fusion experiments as well as in the cytosol (Dixon et al., 2009)

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This pattern suggests that MMN enhancement may precede changes to indices of clinical severity, highlighting the possible utility AEPs as a biomarker of treatment efficacy

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