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This imbalance may contribute to neuroinflammation, mitochondrial dysfunction, and impaired brain developmentall of which are linked to ASD symptoms

As BALB/c mice are melatonin deficient but receptor competent (28), this model allows for the assessment of receptor-mediated versus receptor-independent effects of exogenous melatonin administration in the context of ILC2-driven allergic airways disease

Dou et al., 2018

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